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It has been proposed that the activation of the receptor by ligand binding recruits SHP1 to the JAK2–receptor complex and results in the phosphorylation of the C-terminus of SHP1 where the phosphorylated residues act as docking sites for the Grab2/SOCS1 complex to bind (147)
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Moreover, neurons obtain glucose via the pentose phosphate pathway, emphasizing the complementary metabolic roles of these cell types
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