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Other experimental models of inflammation employing mice are the demonstration of the anti-inflammatory potential of sulforaphane in lung diseases using cigarette smoke-induced alveolar damage [66], cigarette smoke-exposed asthmatic mice [170], bleomycin-induced pulmonary fibrosis [171], ovalbumin (OVA)-sensitised and cigarette smoke-induced airway inflammation [172], chlorine-induced airway hyper-responsiveness [173], haemorrhagic shock-induced lung injury [174], OVA-induced chronic allergic airways [175], lipopolysaccharide (LPS)-induced acute lung injury [176], respiratory syncytial virus (RSV)-induced bronchopulmonary inflammation [177], and the pulmonary arterial hypertension model [178]