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Keywords: Alcohol dependence, alcohol and other drug (AOD) dependence (AODD), addiction, neurobiology, neuroplasticity, neuroadaptation, brain, craving, withdrawal, relapse, neurotransmission, neurotransmitters, glutamate, glutamate receptors, glutamate systems, γ–aminobutyric acid (GABA), GABA systems, dopamine, serotonin, signaling molecules, endogenous opioids, opioid systems, corticotrophin-releasing factor (CRF), animal models, human studies The development of dependence on alcohol (as well as on other drugs of abuse) is posited to involve changes in brain chemistry that lead not only to signs of withdrawal upon abstention from alcohol (i.e., to physical or physiological dependence) (Ritzmann and Tabakoff 1976) but also, in humans, to the behaviors that define alcohol dependence, as described in the most recent edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM–IV) 1 (American Psychiatric Association 1994)